Access the latest scientific research and medical evidence related to Atrophy treatment, diagnosis and symptoms, quickly and easily with doctorAsyou.
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All Pubmed data courtesy of the U.S. National Library of Medicine. Library last updated 18 December 2020.
Clinical trials databases last updated 1 December 2020 (clinicaltrials.gov) and 1 December 2020 (ictrp)
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11512 Abstracts
Research
Propionic Acid Shapes the Multiple Sclerosis Disease Course by an Immunomodulatory Mechanism.
Duscha Alexander, Gisevius Barbara, Hirschberg Sarah, Yissachar Nissan, Stangl Gabriele I ...
Cell Vol 180(6)
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Date 19 Mar 2020
Research
Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy.
Sandri Marco, Sandri Claudia, Gilbert Alex, Skurk Carsten, Calabria Elisa ...
Cell Vol 117(3)
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Date 30 Apr 2004
Research
Myogenin and class II HDACs control neurogenic muscle atrophy by inducing E3 ubiquitin ligases.
Moresi Viviana, Williams Andrew H, Meadows Eric, Flynn Jesse M, Potthoff Matthew J ...
Cell Vol 143(1)
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Date 01 Oct 2010
Research
Tumor induction and tissue atrophy in mice lacking E2F-1.
Yamasaki L, Jacks T, Bronson R, Goillot E, Harlow E ...
Cell Vol 85(4)
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Date 17 May 1996
Research
Identification and characterization of a spinal muscular atrophy-determining gene.
Lefebvre S, Bürglen L, Reboullet S, Clermont O, Burlet P ...
Cell Vol 80(1)
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Date 13 Jan 1995
Research
A novel function for SMN, the spinal muscular atrophy disease gene product, in pre-mRNA splicing.
Pellizzoni L, Kataoka N, Charroux B, Dreyfuss G
Cell Vol 95(5)
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Date 25 Nov 1998
Research
The spinal muscular atrophy disease gene product, SMN, and its associated protein SIP1 are in a complex with spliceosomal snRNP proteins.
Liu Q, Fischer U, Wang F, Dreyfuss G
Cell Vol 90(6)
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Date 19 Sep 1997
Research
The gene for neuronal apoptosis inhibitory protein is partially deleted in individuals with spinal muscular atrophy.
Roy N, Mahadevan M S, McLean M, Shutler G, Yaraghi Z ...
Cell Vol 80(1)
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Date 13 Jan 1995
Research
Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: a mouse model of amyotrophic lateral sclerosis.
Côté F, Collard J F, Julien J P
Cell Vol 73(1)
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Date 09 Apr 1993
Research
Increased expression of neurofilament subunit NF-L produces morphological alterations that resemble the pathology of human motor neuron disease.
Xu Z, Cork L C, Griffin J W, Cleveland D W
Cell Vol 73(1)
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Date 09 Apr 1993
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